Search results for "Model organisms"

showing 10 items of 131 documents

Heat shock response in yeast involver changes in both transcription rates and mRNA stabilities

2011

We have analyzed the heat stress response in the yeast Saccharomyces cerevisiae by determining mRNA levels and transcription rates for the whole transcriptome after a shift from 25uC to 37uC. Using an established mathematical algorithm, theoretical mRNA decay rates have also been calculated from the experimental data. We have verified the mathematical predictions for selected genes by determining their mRNA decay rates at different times during heat stress response using the regulatable tetO promoter. This study indicates that the yeast response to heat shock is not only due to changes in transcription rates, but also to changes in the mRNA stabilities. mRNA stability is affected in 62% of …

Llevat de cervesaTranscription GeneticEstrès oxidatiuRNA StabilitySaccharomyces cerevisiaeGene Expressionlcsh:MedicineYeast and Fungal ModelsRNA-binding proteinSaccharomyces cerevisiaeModels BiologicalGenètica molecularModel OrganismsTranscripció genèticaGenome Analysis ToolsTranscription (biology)Gene Expression Regulation FungalYeastsHeat shock proteinMolecular Cell BiologyGeneticsCluster AnalysisRNA MessengerHeat shocklcsh:ScienceBiologyGeneTranscription factorHeat-Shock ProteinsMultidisciplinaryBase SequenceOrganisms Genetically ModifiedbiologySystems Biologylcsh:RRNA FungalLlevats -- GenèticaGenomicsbiology.organism_classificationMolecular biologyFunctional GenomicsCell biologyRegulonRNAlcsh:QGenome Expression AnalysisHeat-Shock ResponseResearch ArticleTranscription Factors
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Mismatch negativity (MMN) in freely-moving rats with several experimental controls.

2014

Mismatch negativity (MMN) is a scalp-recorded electrical potential that occurs in humans in response to an auditory stimulus that defies previously established patterns of regularity. MMN amplitude is reduced in people with schizophrenia. In this study, we aimed to develop a robust and replicable rat model of MMN, as a platform for a more thorough understanding of the neurobiology underlying MMN. One of the major concerns for animal models of MMN is whether the rodent brain is capable of producing a human-like MMN, which is not a consequence of neural adaptation to repetitive stimuli. We therefore tested several methods that have been used to control for adaptation and differential exogenou…

lcsh:MedicineMismatch negativityNeurophysiologyBiologyStimulus (physiology)ElectroencephalographyAuditory cortexResearch and Analysis Methodsbehavioral disciplines and activitiesModel OrganismsEvent-related potentialmedicineAnimalsHumanslcsh:ScienceOddball paradigmta515Auditory CortexBrain MappingMultidisciplinarymedicine.diagnostic_testlcsh:RNeural adaptationSkullBiology and Life SciencesElectroencephalographyAnimal ModelsSensory SystemsFrontal LobeRatsmedicine.anatomical_structureFrontal lobeAcoustic StimulationBrain ElectrophysiologyAuditory SystemModels AnimalEvoked Potentials Auditorylcsh:QNeuroscienceResearch ArticleNeurosciencePloS one
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Candida albicans-epithelial interactions: dissecting the roles of active penetration, induced endocytosis and host factors on the infection process

2012

International audience; Candida albicans frequently causes superficial infections by invading and damaging epithelial cells, but may also cause systemic infections by penetrating through epithelial barriers. C. albicans is a remarkable pathogen because it can invade epithelial cells via two distinct mechanisms: induced endocytosis, analogous to facultative intracellular enteropathogenic bacteria, and active penetration, similar to plant pathogenic fungi. Here we investigated the contributions of the two invasion routes of C. albicans to epithelial invasion. Using selective cellular inhibition approaches and differential fluorescence microscopy, we demonstrate that induced endocytosis contri…

[SDV]Life Sciences [q-bio]lcsh:MedicineYeast and Fungal ModelsPathogenesisCandidiasis OralMolecular Cell BiologyCandida albicanslcsh:ScienceCandida albicansPathogencandida albicans;epithelial interaction;endocytosis;infection0303 health sciencesFungal proteinMultidisciplinaryFungal DiseasesBlood Physiological PhenomenaCadherinsEndocytosisCorpus albicansepithelial interactionCell biologyHost-Pathogen InteractionInfectious Diseases[SDE]Environmental SciencesHost-Pathogen InteractionsMedicineCellular TypesSuperficial MycosesCandidalysinResearch ArticleMycologyBiologyEndocytosisMicrobiologyCell LineMicrobiologyFungal Proteins03 medical and health sciencesModel OrganismsMicroscopy Electron TransmissionCell Adhesion[SDV.BV]Life Sciences [q-bio]/Vegetal BiologyHumansCell adhesionBiology030304 developmental biology030306 microbiologyIntracellular parasitelcsh:RFungiMouth MucosaEpithelial Cellsbiology.organism_classificationinfectionYeastlcsh:Q
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A luminal glycoprotein drives dose-dependent diameter expansion of the Drosophila melanogaster hindgut tube

2012

An important step in epithelial organ development is size maturation of the organ lumen to attain correct dimensions. Here we show that the regulated expression of Tenectin (Tnc) is critical to shape the Drosophila melanogaster hindgut tube. Tnc is a secreted protein that fills the embryonic hindgut lumen during tube diameter expansion. Inside the lumen, Tnc contributes to detectable O-Glycans and forms a dense striated matrix. Loss of tnc causes a narrow hindgut tube, while Tnc over-expression drives tube dilation in a dose-dependent manner. Cellular analyses show that luminal accumulation of Tnc causes an increase in inner and outer tube diameter, and cell flattening within the tube wall,…

glycoproteinCancer ResearchhindgutOrganogenesis[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritiontenectinHydrostatic pressureExtracellular matrixlumenMolecular Cell BiologyMorphogenesisDrosophila Proteinslumen;hindgut;tenectin;epithelial tube;glycoproteinGenetics (clinical)Animal biologyExtracellular Matrix ProteinsDrosophila MelanogasterGene Expression Regulation DevelopmentalHindgutAnimal ModelsAnatomymusculoskeletal systemExtracellular MatrixCell biologymedicine.anatomical_structureAlimentation et NutritionResearch Articleepithelial tubelcsh:QH426-470MorphogenesisLumen (anatomy)BiologyModel OrganismsGenetic MutationBiologie animaleGeneticsmedicineAnimalsFood and NutritionBiologyMolecular BiologyEcology Evolution Behavior and SystematicsGlycoproteinsEmbryonic stem cellExtracellular Matrix CompositionEpitheliumGastrointestinal Tractlcsh:GeneticsMutagenesisEctopic expressionGene Function[SDV.AEN]Life Sciences [q-bio]/Food and NutritionOrganism DevelopmentDevelopmental Biology
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Mechanical ventilation alters the development of staphylococcus aureus pneumonia in rabbit

2016

Ventilator-associated pneumonia (VAP) is common during mechanical ventilation (MV). Beside obvious deleterious effects on muco-ciliary clearance, MV could adversely shift the host immune response towards a pro-inflammatory pattern through toll-like receptor (TLRs) up-regulation. We tested this hypothesis in a rabbit model of Staphylococcus aureus VAP. Pneumonia was caused by airway challenge with S. aureus, in either spontaneously breathing (SB) or MV rabbits (n = 13 and 17, respectively). Pneumonia assessment regarding pulmonary and systemic bacterial burden, as well as inflammatory response was done 8 and 24 hours after S. aureus challenge. In addition, ex vivo stimulations of whole blood…

0301 basic medicinePulmonologyPhysiologyStaphylococcusmedicine.medical_treatment[SDV]Life Sciences [q-bio]lcsh:MedicinePharmacologyPathology and Laboratory Medicinemedicine.disease_causeStaphylococcal/immunology/pathologyImmune ReceptorsBiochemistry0302 clinical medicineImmune PhysiologyPneumonia StaphylococcalMedicine and Health SciencesMedicineStaphylococcus Aureuslcsh:ScienceImmune ResponseToll-like ReceptorsMammalsddc:616Innate Immune SystemImmune System ProteinsMultidisciplinaryddc:617RespirationPneumonia Ventilator-AssociatedInterleukinAnimal ModelsHematologyBacterial PathogensBody Fluids3. Good healthBloodmedicine.anatomical_structureMedical MicrobiologyStaphylococcus aureusVertebratesArtificialCytokinesRabbitsPathogensAnatomymedicine.symptomStaphylococcus aureus/immunologyResearch ArticleSignal TransductionToll-Like Receptor 2/immunologyImmunologyInflammationLung injuryResearch and Analysis MethodsMicrobiology03 medical and health sciencesModel OrganismsSigns and SymptomsDiagnostic MedicineAnimalsMicrobial PathogensInflammationMechanical ventilationInterleukin-8/immunologyLung[ SDV ] Life Sciences [q-bio]BacteriaTumor Necrosis Factor-alphabusiness.industrylcsh:RInterleukin-8OrganismsBiology and Life SciencesProteins030208 emergency & critical care medicineCell BiologyPneumoniaMolecular Developmentmedicine.diseaseTumor Necrosis Factor-alpha/immunologyRespiration ArtificialToll-Like Receptor 2Pneumonia030104 developmental biologyVentilator-Associated/immunology/microbiology/pathologyImmune SystemAmniotesImmunologylcsh:QbusinessSpleenEx vivoDevelopmental Biology
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Transcriptional Differences between Diapausing and Non-Diapausing D. montana Females Reared under the Same Photoperiod and Temperature

2016

Background A wide range of insects living at higher latitudes enter diapause at the end of the warm season, which increases their chances of survival through harsh winter conditions. In this study we used RNA sequencing to identify genes involved in adult reproductive diapause in a northern fly species, Drosophila montana. Both diapausing and non-diapausing flies were reared under a critical day length and temperature, where about half of the emerging females enter diapause enabling us to eliminate the effects of varying environmental conditions on gene expression patterns of the two types of female flies. Results RNA sequencing revealed large differences between gene expression patterns of…

0301 basic medicinePhysiologyMolecular biologylcsh:MedicineDiapause InsectBiochemistryTranscriptomeSequencing techniquesCytochrome P-450 Enzyme SystemGlucose MetabolismLääketieteen bioteknologia - Medical biotechnologyGene expressionMedicine and Health SciencesDrosophila Proteinsgeeniekspressiolcsh:SciencegenesOverwinteringGeneticsMultidisciplinaryBiolääketieteet – BiomedicinebiologyReproductionDrosophila MelanogasterMetamorphosis BiologicalTemperatureInsect physiologyRNA sequencingAnimal ModelsGenomicsPhenotypeOvariesInsectsCarbohydrate MetabolismDrosophilaFemaleAnatomyDrosophila melanogasterTranscriptome AnalysisResearch ArticleArthropodaPhotoperiodMyosinsDiapause03 medical and health sciencesExtraction techniquesModel OrganismsDrosophila montanaGeneticsAnimalsGenegeenitta1184lcsh:RReproductive SystemOrganismsBiology and Life SciencesComputational BiologyGenome Analysisbiology.organism_classificationInvertebratesActinsRNA extractionResearch and analysis methodsdiapauseMolecular biology techniquesMetabolism030104 developmental biologygene expressionta1181lcsh:QPhysiological ProcessesDevelopmental BiologyPLoS ONE
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''Circulating and Brain BDNF Levels in Stroke Rats. Relevance to Clinical Studies''

2011

International audience; ''Background: Whereas brain-derived neurotrophic factor (BDNF) levels are measured in the brain in animal models of stroke, neurotrophin levels in stroke patients are measured in plasma or serum samples. The present study was designed to investigate the meaning of circulating BDNF levels in stroke patients. Methods and Results: Unilateral ischemic stroke was induced in rats by the injection of various numbers of microspheres into the carotid circulation in order to mimic the different degrees of stroke severity observed in stroke patients. Blood was serially collected from the jugular vein before and after (4 h, 24 h and 8 d) embolization and the whole brains were co…

Malelcsh:MedicineEDEMA''CardiovascularBiochemistry0302 clinical medicineNeurotrophic factorsJugular veinEdemaBlood plasmaNeurobiology of Disease and RegenerationMedicineHippocampus (mythology)Plateletlcsh:ScienceStroke0303 health sciencesMultidisciplinary''NEUROTROPHIC FACTORbiologyBrainNeurochemistryAnimal ModelsRECOVERYDEPRESSIONPLATELETSStrokeISCHEMIC-STROKENeurologyOrgan SpecificityAnesthesia[ SCCO.NEUR ] Cognitive science/NeuroscienceMedicinemedicine.symptomNeurotrophinResearch Articlemedicine.medical_specialtyCerebrovascular DiseasesImmunologyMETABOLISM03 medical and health sciencesModel OrganismsInternal medicineAnimalsHumanscardiovascular diseasesRats WistarImmunoassaysBiology030304 developmental biologyIschemic Strokebusiness.industryBrain-Derived Neurotrophic Factor[SCCO.NEUR]Cognitive science/Neurosciencelcsh:REDEMAmedicine.diseaseENDOTHELIAL-CELLSRatsEndocrinologynervous systemMARKERbiology.proteinImmunologic TechniquesRatlcsh:QClinical Immunologybusiness030217 neurology & neurosurgeryNeuroscienceNEUROTROPHIC FACTOR
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Compromised Neurotrophic and Angiogenic Regenerative Capability during Tendon Healing in a Rat Model of Type-II Diabetes

2017

Metabolic diseases such as diabetes mellitus type-II (DM-II) may increase the risk of suffering painful connective tissue disorders and tendon ruptures. The pathomechanisms, however, by which diabetes adversely affects connective tissue matrix metabolism and regeneration, still need better definition. Our aim was to study the effect of DM-II on expressional changes of neuro- and angiotrophic mediators and receptors in intact and healing Achilles tendon. The right Achilles tendon was transected in 5 male DM-II Goto-Kakizaki (GK) and 4 age-matched Wistar control rats. The left Achilles tendons were left intact. At week 2 post-injury, NGF, BDNF, TSP, and receptors TrkA, TrkB and Nk1 gene expre…

Male0301 basic medicinePhysiologyGene Expressionlcsh:MedicineSubstance PCardiovascular PhysiologyTendonsEndocrinology0302 clinical medicineNerve Growth FactorMedicine and Health SciencesHomeostasisMedicinelcsh:ScienceMammalsAchilles tendonMultidisciplinarybiologyAnimal ModelsAnatomyReceptors Neurokinin-1musculoskeletal systemTendonmedicine.anatomical_structureExperimental Organism SystemsConnective TissueVertebratesAnatomyResearch ArticleNeurotrophinmedicine.medical_specialtyWistar RatsEndocrine DisordersNeovascularization PhysiologicConnective tissueResearch and Analysis MethodsRodentsAchilles Tendon03 medical and health sciencesModel OrganismsTendon InjuriesInternal medicineTissue RepairDiabetes MellitusGeneticsAnimalsReceptor trkBRats WistarReceptor trkABrain-derived neurotrophic factorWound Healingbusiness.industryBrain-Derived Neurotrophic Factorlcsh:RScleraxisOrganismsBiology and Life SciencesRatsTenomodulinDisease Models AnimalBiological Tissue030104 developmental biologyNerve growth factorEndocrinologyDiabetes Mellitus Type 2Metabolic DisordersAmniotesbiology.proteinlcsh:QAngiogenesisPhysiological Processesbusiness030217 neurology & neurosurgeryDevelopmental BiologyPLOS ONE
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Volatile Anesthetics Influence Blood-Brain Barrier Integrity by Modulation of Tight Junction Protein Expression in Traumatic Brain Injury

2012

Disruption of the blood-brain barrier (BBB) results in cerebral edema formation, which is a major cause for high mortality after traumatic brain injury (TBI). As anesthetic care is mandatory in patients suffering from severe TBI it may be important to elucidate the effect of different anesthetics on cerebral edema formation. Tight junction proteins (TJ) such as zonula occludens-1 (ZO-1) and claudin-5 (cl5) play a central role for BBB stability. First, the influence of the volatile anesthetics sevoflurane and isoflurane on in-vitro BBB integrity was investigated by quantification of the electrical resistance (TEER) in murine brain endothelial monolayers and neurovascular co-cultures of the B…

MaleMouse610 MedizinBrain EdemaPharmacologyCardiovascularMiceAnesthesiology610 Medical sciencesEdemaMolecular Cell BiologyClaudin-5MultidisciplinaryIsofluraneQRAnimal ModelsHead Injurymedicine.anatomical_structureNeurologyBlood-Brain BarrierAnesthesiaAnesthetics InhalationMedicineCellular Typesmedicine.symptomResearch Articlemedicine.drugMethyl EthersTraumatic brain injuryCerebrovascular DiseasesScienceBrain damageBlood–brain barrierSevofluraneCell LineTight JunctionsCerebral edemaSevofluraneModel OrganismsVascular BiologymedicineAnimalsBiologybusiness.industryEndothelial Cellsmedicine.diseaseCoculture TechniquesIsofluraneBrain InjuriesAnestheticZonula Occludens-1 ProteinMolecular NeurosciencebusinessNeurosciencePLoS ONE
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Correction: Recovery from Toxic-Induced Demyelination Does Not Require the NG2 Proteoglycan.

2018

[This corrects the article DOI: 10.1371/journal.pone.0163841.].

ImmunologyGene ExpressionMouse ModelsCell MigrationResearch and Analysis MethodsPathology and Laboratory MedicineCorpus CallosumDirected Cell MigrationModel OrganismsNerve FibersSigns and SymptomsAnimal CellsDiagnostic MedicineMedicine and Health SciencesGeneticsImmune ResponseNeuronsInflammationChemotaxisBiology and Life SciencesBrainCell DifferentiationAnimal ModelsCell BiologyAxonsCell MotilityCellular NeuroscienceCellular TypesAnatomyResearch ArticleDevelopmental BiologyNeurosciencePloS one
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